I guess I'm not understanding the mechanism for success. If the liposomes sequester just the toxins, how is the infection itself eliminated? Is the idea that staving off the deadly toxins buys the host enough time to catch up to and effectively fight the infection?
Also their claim of not promoting bacterial resistance reeks of bogosity if there's any selective pressure put on the bacteria. That pressure--and it sure looks like it's there to me--will cause the bacteria to evolve a resistance/workaround for this treatment as well.